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This pathway was originally characterized in nerve cells by our laboratory and named oxytosis, a process initiated by inhibition of the cystine/glutamate antiporter, leading to GSH depletion, accumulation of reactive oxygen species, and lethal lipid peroxidation [17, 18]

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ROS-induced damage to DNA, proteins, and lipids can directly lead to cell death, but it also promotes microglial activation and the release of pro-inflammatory cytokines, further exacerbating neurodegeneration (Tfilin et al., 2023)