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There is a cross-tolerance between alcohol, benzodiazepines, barbiturates, nonbenzodiazepine drugs, and corticosteroids, which all act by enhancing the GABAA receptor’s function via modulating the chloride ion channel function of the GABA A receptor [233,234,235,236]

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Unlike endogenous gastric proteins, BPC-157 is stable under physiological conditions and resistant to enzymatic degradation in the gastrointestinal tract, making it suitable for research across both systemic and local tissue-repair models