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In phenylketonuria, altered amino acid metabolism leads to deficiencies in neurotransmitters like serotonin, dopamine, and norepinephrine, resulting in delayed sleep onset, prolonged latency, and excessive daytime sleepiness (Muriach et al., 2014

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Of note, all of these disturbances were consistently attenuated with the application of BPC 157 therapy and activation of the collateral pathways, relayed on the given injury [11,12,13,14,15,16,17,18,19,20,21,22,23,24,25]