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The cuprizone model is extensively used to study in vivo toxicity-induced demyelination (Zhang et al., 2017a) with less infiltrated immune cells as compared to what found in EAE (Procaccini et al., 2015)

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14 - 21 Consequently, as we emphasized, 4 since the original organoprotective/healing intention of stress coping response 22 confronts with unsuccessful realization by the Selye’s “first mediator” in stress response, 25 that integrative mediator that integrates the adaptive bodily response to stress, remains obscured in the highly sophisticated latter Selye’s stress concept presentation